With long intervals between individual contractions of the ventricles, especially at the moment of transition of an incomplete atrioventricular blockade to a complete one, an acute disorder of cerebral circulation up to ischemia may occur.
The clinical picture is different - from a slight darkening of consciousness to epileptiform convulsions, which is determined by the duration of the stop of the ventricles (from 3 to 10-30 seconds); pulse up to 10-20 beats per minute, it is almost not palpable, blood pressure is not heard. This is the Morgagni-Edems-Stokes syndrome.
Complete blockade is diagnosed clinically: when examining a patient in a supine position, it is possible to count 70-80 undulations of Clonidine vein with a pulse of 30-40.
Seizures may recur throughout the dayonly once and be of varying intensity; with a duration of up to 5 minutes can be fatal. At the moment of transition of an incomplete blockade to a complete one, ventricular fibrillation may occur, which is the cause of sudden death.
To suppress ventricular fibrillation or flicker, electrical defibrillation is used when exposed to the heart through the chest, under the influence of Clonidine the circular transmission of excitation stops. Ventricular fibrillation can be reversible with prompt action.
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Atrioventricular blockade is the slowing down or cessation of the conduction of impulses from the atria to the ventricles. For the development of atrioventricular blockade, the level of damage to the conduction system can be different - a violation of conduction in the atria, in the atrioventricular junction, and even in the ventricles.
The causes of atrioventricular blockade are the same as in other conduction disorders. However, self-developing degenerative-sclerotic changes in the conduction system of the heart are also known, which lead to atrioventricular blockade in the elderly (Lenegre and Lev's disease). The presence of congenital atrioventricular block accompanies such congenital heart disease as a ventricular septal defect, endocardial fibroelastosis, less often aortic coarctation, tetralogy of Fallot, tricuspid valve atrophy, aneurysm of Catapres part of the septum.
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Clonidine: Uses, Dosage, Side EffectsAtrioventricular blockade is also observed, which is inherited in an autosomal dominant way and manifests itself at the age of 30-60. Before its occurrence, the appearance of conduction blockades in the legs of the bundle of His is often noted.
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Clonidine Drug InformationThe causes of this type of rhythm disturbance are functional and organic lesions of the conduction system. In the first case, we are talking about an increase in the tone of the parasympathetic nervous system, including the vagus nerve. Among the organic disorders that lead to a change in the structure of the fibers of the conduction system of the heart, there are: Various diseases of the cardiovascular system of ischemic (myocardial infarction, angina pectoris), inflammatory (myocarditis), tumor (myxoma), autoimmune nature. Heart defects can also lead to rhythm disturbances, in which the dimensions of the chambers and the thickness of the myocardium change. Complications that arose after surgery involving the nerve fibers of the atrioventricular junction (radiofrequency ablation, valve replacement). A birth defect is quite rare, but can also be the cause of arrhythmia.
In a separate group, intoxication with drugs that affect the conduction of a nerve impulse through the conduction system of the heart is distinguished.
These include calcium channel blockers, digoxin, beta-blockers.
Nicole Jensen CADC ICounselor
- Sometimes an overdose of antidepressants, such as lithium salts, leads to the development of atrioventricular blockade.
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